Description
Book SynopsisThere are three major types of human retroviruses, namely HIV, HTLV, and endogenous human retroviruses. This book presents the latest findings on the replication of these human retroviruses. This book is unique in that there has been no comparable book that integrates the findings from the three known classes of human retroviruses. Other books have focused on one of the three classes of human retroviruses individually.An accomplished international team of contributing authors have combined their expertise to provide cutting-edge findings in this important field. The book will be a valuable reference for students, researchers and medical professionals.
Table of ContentsThe Role of Hematopoietic Progenitor Cells in Retroviral Pathogenesis (P Banerjee et al.); Human T Cell Leukemia Virus Type 1, Cellular Transformation, and Adult T Cell Leukemia (J Yasunaga & K-T Jeang); Application of Proteomics to HTLV-1: Understanding Pathogenesis and Enhancing Diagnostics (O J Semmes et al.); Antisense Transcription in Human T-Cell Leukemia Virus Type 1: Discovery of a New Viral Gene (B Barbeau et al.); Regulation of HTLV-1 Transcription by Viral and Cellular Proteins (N Polakowski & I Lemasson); Cellular Factors Involved in HIV-1 RNA Transport (C Williams et al.); Integration Site Selection by Retroviruses and Retroviral Vectors (C Cattoglio & F Mavilio); Syncytins in Normal and Pathological Placentas (A Malassine et al.); Vpu, Tetherin and Innate Immunity: Antiviral Restriction of Retroviral Particle Release (S J D Neil); Retrovirus Replication: New Perspectives on Enzyme and Substrate Dynamics (J W Rausch et al.); Non-Human Primates in HIV-1 Research (W M J M Bogers); Lentiviral Integration and the Role of the Cellular Cofactors LEDGF/P75 and Transportin-SR2 (B Van Heertum et al.); Retrovirus Restriction Factors (R S Harris et al.); Rev Revisited: Additional Functions of the HIV-1 Rev Protein (B Grewe & K A berla); HIV-1 Interactions with Small RNA Induced Silencing Mechanisms (J Haasnoot & B Berkhout);
