Description
Book SynopsisNeuropathology of Transmissible Spongiform Encephalopathies (Prion Diseases).- Central Pathogenesis of Prion Diseases.- Hereditary Prion Protein Amyloidoses.- Mouse Behavioural Studies and What They Can Teach Us about Prion Diseases.- Electrophysiological Approaches to the Study of Prion Diseases.- Prion Protein, Prion Protein-Like Protein, and Neurodegeneration.- Oxidative Stress and Mitochondrial Dysfunction in Neurodegeneration of Transmissible Spongiform Encephalopathies (TSEs).- Mechanisms of Prion Toxicity and Their Relationship to Prion Infectivity.- A Stone Guest on the Brain: Death as a Prion.- Molecular Mechanisms Mediating Neuronal Cell Death in Experimental Models of Prion Diseases, in vitro.- Processing and Mis-Processing of the Prion Protein: Insights into the Pathogenesis of Familial Prion Disorders.- Signaling Pathways Controling Prion Neurotoxicity: Role of Endoplasmic Reticulum Stress-Mediated Apoptosis.- Cell Culture Models to Unravel Prion Protein Function and Aberr
Table of ContentsNeuropathology of transmissible spongiform encephalopathies (prion diseases).- Central pathogenesis of prion diseases.- Hereditory prion protein Amyloidoses.- Mouse behavioural studies and what they can teach us about prion diseases.- Electrophysiological approaches to the study of prion diseases.- Prion protein, prion protein-like protein, and neurodegeneration.- Oxidative stress and mitochondrial dysfunction in neurodegeneration of transmissible spongiform encephalopathies (TSEs).- Mechanisms of prion toxicity and their relationship to prion infectivity.- A stone guest on the brain: Death as a prion.- Molecular mechanisms mediating neuronal cell death in experimental models of prion diseases, in vitro.- Processing and mis-processing of the prion protein: Insights into the pathogenesis of familial prion disorders.- Signaling pathways controling prion protein neurotoxicity: Role of endoplasmic reticulum stress-mediated apoptosis.- Cell culture models to unravel prion protein function and aberrancies in TSE.- Insights into the cellular trafficking of prion proteins.- The molecular basis of prion protein-mediated neuronal damage.
