{"product_id":"the-hippocampus-volume-4-9781461580263","title":"The Hippocampus Volume 4","description":"\u003cb\u003eBook Synopsis\u003c\/b\u003e\u003cbr\u003eThe current volumes of The Hippocampus reflect the prodigious amount of work aimed at discovering the functions of this structure over the past decade.\u003cbr\u003e\u003cbr\u003e\u003cb\u003eTable of Contents\u003c\/b\u003e\u003cbr\u003e1. An Executive Function of the Hippocampus: Pathway Selection for Thalamic Neuronal Significance Code.- 1. Introduction.- 1.1. Prologue.- 1.2. Working Assumptions.- 2. Methods: Discrete Lesions and Recording of Unit Activity during Discriminative Avoidance Behavior.- 2.1. Behavioral Training.- 2.2. Basic Tactics of the Approach.- 3. A Working Model for Limbic Telodiencephalic Function.- 3.1. Anatomical Focus.- 3.2. Involvement of Nonthalamic Projection Targets of the Cingulate Cortex in Contextual Priming of Learned Behaviors.- 3.3. Thalamic Nuclei as the Primary Sources of Associative CS-Elicited Neuronal Excitation of Cingulate Cortical Neurons.- 3.4. Elicitation of Primed Behavioral Responses by CS-Elicited Volleys of Thalamic Origin Projected through the Cingulate Cortex.- 3.5. Cingulate Cortical Limiting of Thalamic Activity and Behavior.- 3.6. Hippocampal Role in Pathway Selection.- 3.7. Interaction of Behavior-Limiting and -Inducing Pathways within Trials.- 3.8. Hippocampal Comparison Processes and Pathway Selection.- 4. Supporting Data.- 4.1. The AVN and the MDN as Sources of CS-Elicited Excitatory Drive of Cingulate Cortical Synaptic Circuitry.- 4.2. Cingulate Cortical Limiting of the AVN.- 4.3. Hippocampal Selection of the Limiting Pathway.- 4.4. Data Supporting the Model’s Attributions of Behavioral Relevance to the Limbic Structures.- 4.5. Contextual Stimuli, Comparison Processes, and Priming.- 4.6. Brief Recapitulation.- 5. Implications for Human Memory Mechanisms.- 5.1. Neuropathology of Amnesia.- 5.2. Intact Functions.- 6. References.- 2. Brainstem Modulations of the Hippocampus: Anatomy, Physiology, and Significance.- 1. Introduction.- 2. Mechanisms of Theta Generation within the Hippocampus.- 3. The Cholinergic Septohippocampal Theta-Generating System.- 4. Brainstem Systems Controlling the Hippocampal EEG.- 4.1. Brainstem Stimulation Studies.- 5. Unit Activity of Brainstem Nuclei Implicated in the Generation of the Theta Rhythm.- 5.1. Pontine Reticular Formation.- 5.2. Midbrain RF, Locus Coeruleus, and Median Raphe.- 6. Ascending Brainstem-to-Septohippocampal Pathways Involved in the Control of the Hippocampal EEG.- 6.1. Stimulation Studies.- 6.2. Anatomical Studies.- 7. Significance of the Hippocampal Theta Rhythm.- 7.1. Septal Potentiation of Hippocampal Population Responses at CAI and the Dentate Gyrus.- 7.2. Potentiation of Hippocampal Population Responses during Theta-Associated Behaviors in Freely Moving Animals.- 7.3. A Possible Role for Theta in Long-Term Potentiation and Memory.- 8. General Summary and Conclusions.- 9. References.- 3. Behaviorally Dependent Neuronal Gating in the Hippocampus.- 1. Neuronal Gating.- 2. Brainstem Influences on Neuronal Gating.- 2.1. Serotonin.- 2.2. Reticular Formation.- 2.3. Norepinephrine.- 3. Discussion.- 4. References.- 4. Selective Hippocampal Lesions and Behavior: Implications for Current Research and Theorizing.- 1. Introduction.- 2. Neuroanatomical Considerations.- 3. Early Research Using Lesions Limited to Hippocampal Subregions.- 4. The Use of Neurotoxins to Selectively Lesion the Hippocampus.- 5. Behavioral Effects of Selective Neurotoxin Lesions.- 5.1. Experiments Designed to Study Hippocampal Involvement in Place versus Cue Learning and Working Memory versus Reference Memory.- 5.2. Other Experiments Involving Selective Neurotoxin Lesions of the Hippocampus.- 6. Implications for Theories of Hippocampal Function and Current Research in the Area.- 7. Summary and Conclusions.- 8. References.- 5. Cholinergic and Catecholaminergic Modification of the Hippocampal Lesion Syndrome.- 1. Early Attempts to Alleviate the Behavioral Impairments by External Manipulations.- 2. Secondary Neurochemical Changes Following Hippocampal Damage.- 3. Cholinergie Manipulations and Hippocampal Lesion Effects.- 4. Glucocorticoid Changes and Hippocampal Lesion Effects.- 5. An Analysis of Hippocampal Effects in the Open Field.- 5.1. Factor 1.- 5.2. Factor 2.- 5.3. Factor 3.- 5.4. Factor 4.- 6. Spontaneous Alternation.- 7. Conclusions.- 8. References.- 6. Comparator and Buffer Memory: An Attempt to Integrate Two Models of Hippocampal Function.- 1. The Hippocampus as a Comparator.- 1.1. Antianxiety Drug Action.- 1.2. Prolegomena to a Theory of Hippocampal Function.- 1.3. Gray’s Theory of Hippocampal Function.- 1.4. Two Ways of Activating the SHS.- 2. The Hippocampus as a Memory Buffer.- 3. Relationship between the Models.- 3.1. Partial Reinforcement Experiments.- 3.2. An Attempted Resolution.- 4. Antianxiety Drugs and the Septhohippocampal System.- 5. Conclusions.- 6. References.- 7. Role of the Hippocampus in Classical Conditioning of Aversive and Appetitive Behaviors.- 1. Introduction.- 2. Hippocampal Unit Activity during Classical Conditioning of the NM and CJM Responses.- 2.1. Pyramidal and Theta Cell Responses during NM Conditioning.- 2.2. Hippocampal Unit Responses during CJM Conditioning.- 2.3. Dentate Granule Cell Responses during NM Conditioning.- 2.4. Hippocampal Slow Waves and NM Conditioning.- 3. Intrinsic Changes in the Hippocampus as a Result of Classical Conditioning: Evidence and Potential Mechanisms.- 4. Relationship between Hippocampal Neuronal Activity and Learned Behavior 22..- 4.1. Correlations between Hippocampal Unit Activity and Conditioned Behavior.- 4.2. Necessity of the Hippocampus for Complex but Not Simple Classical Conditioning Paradigms.- 4.3. Role of the Hippocampus in Delay Conditioning.- 4.4. Relationship of Hippocampal Neuronal Plasticity to Motoneuronal Activity Responsible for NM and CJM Movement.- 5. Multisynaptic Anatomical Pathways Underlying the Behavioral Consequences of Hippocampal Neuronal Plasticity.- 5.1. Projections to Retrohippocampal Brain Regions.- 5.2. Subcortical Targets.- 6. Summary.- 7. References.- 8. The Monkey and the Sea Horse.- 1. Introduction.- 2. Behavioral Profile of Adult Monkeys with Damage to the Hippocampal System.- 2.1. Bilateral Ablations of the Hippocampal Formation.- 2.2. Bilateral Sections of the Fornix.- 3. Effects of Hippocampal Damage as a Function of Age of Lesion.- 3.1. Subjects.- 3.2. Surgery and Histological Verification of Lesions.- 3.3. Methods.- 3.4. Results.- 4. Associative Learning Spared, Recognition and Associative Memory Not Spared, after Early Ablations of Hippocampus.- 4.1. Concurrent Object Discrimination Learning.- 4.2. Recognition Memory.- 4.3. Object Recognition Task: Delays.- 4.4. Object Recognition Task: Lists.- 4.5. Association Memory Task.- 4.6. Position Recognition Memory.- 5. Summary and Comments.- 6. Conclusions.- 7. References.- 9. Hippocampal Function and Memory for Temporal Context.- 1. Introduction.- 1.1. The Hippocampal System.- 1.2. Rats as an Animal Model.- 2. Comparative Cognition.- 2.1. Comparisons between Species.- 2.2. Comparisons through Lesion Analysis.- 3. Memory for the Temporal Context of an Event.- 4. Animal Tests of Working and Reference Memory.- 5. Memory for Temporal Intervals.- 6. Conclusions.- 7. References.- 10. Memory for Unique Instances: Temporal Lobe and Hippocampal Dependence.- 1. The Working Memory Model of Temporal Lobe Deficits.- 2. A New Animal Model of Temporal Lobe Memory Deficits.- 3. A Test of the Model.- 3.1. General Methods.- 3.2. Phase 1 of the Experiment.- 3.3. Phase 2 of the Experiment.- 3.4. Predictions and Results.- 4. Application of the Model to Cognitive Place Learning.- 4.1. Acquisition of Place.- 4.2. Reorientation to Place.- 4.3. Accessing Separate Events at One Place.- 5. The Value of Unique Instance Memories.- 6. Evaluation of the Model.- 6.1. Regulation in Relation to the Facts of a Unique Context.- 6.2. Regulation in Relation to a Recalled Instance.- 7. Conclusions.- 7.1. Retrograde Characteristics.- 7.2. Retroactive and Categorical Characteristics.- 8. References.- 11. The Hippocampal System and Recombinant Processing.- 1. Introduction.- 1.1. The Enigma.- 2. Limbic versus Nonlimbic Learning and Memory.- 2.1 Alternation versus Discrimination.- 2.2. Henry M..- 2.3. Discrimination Reversal.- 2.4. Effort.- 2.5. Competence.- 3. Hippocampus versus Amygdala: Novelty and Familiarity.- 3.1. Factors That Influence Processing Span.- 3.2. Discrimination Reversal.- 3.3. Learning, Extinction, and Relearning.- 3.4. Paired versus Scattered Stimuli.- 3.5. Massed versus Spaced Trials.- 3.6. Reaction to Novel Stimuli.- 3.7. Errors of Omission and of Commission.- 4. The Hippocampus and Competence.- 4.1. Orienting and Habituation.- 4.2. Alternation versus Delayed Response.- 4.3. Internally versus Externally Ordered Sequences.- 4.4. Spatial versus Nonspatial Tasks.- 4.5. Hippocampal Electrical Activity.- 4.6. Mechanism.- 4.7. Context-Sensitive Recombinant Processing.- 5. Whither Now?.- 5.1. Issues.- 5.2. Disparate Interpretations of Related Databases.- 5.3. Theoretical Frames and the Problem of Conscious Cognizance.- 6. References.","brand":"Springer Us","offers":[{"title":"Default Title","offer_id":51742559535447,"sku":"9781461580263","price":42.74,"currency_code":"GBP","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0817\/1739\/5799\/files\/9781461580263.jpg?v=1758385232","url":"https:\/\/bookcurl.com\/products\/the-hippocampus-volume-4-9781461580263","provider":"Book Curl","version":"1.0","type":"link"}